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MxA overexpression reveals a common genetic link in four Fanconi anemia complementation groups.
Fanconi anemia (FA) consists of a group of at least five autosomal recessive disorders that share both clinical (e.g., birth defects and hematopoietic failure) and cellular (e.g., sensitivity to cross-linking agents and predisposition to apoptosis) features with each other. However, a common pathoge...
Uloženo v:
| Vydáno v: | J Clin Invest |
|---|---|
| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Clinical Investigation
1997
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC508494/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9389754/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI119836 |
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