Slow ventricular conduction in mice heterozygous for a connexin43 null mutation.

To characterize the role of the gap junction protein connexin43 (Cx43) in ventricular conduction, we studied hearts of mice with targeted deletion of the Cx43 gene. Mice homozygous for the Cx43 null mutation (Cx43 -/-) die shortly after birth. Attempts to record electrical activity in neonatal Cx43...

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Bibliografski detalji
Izdano u:J Clin Invest
Glavni autori: Guerrero, P A, Schuessler, R B, Davis, L M, Beyer, E C, Johnson, C M, Yamada, K A, Saffitz, J E
Format: Artigo
Jezik:Inglês
Izdano: American Society for Clinical Investigation 1997
Teme:
Research Article
Online pristup:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC508024/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9109444/
https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI119367
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