IKBKE protein activates Akt independent of phosphatidylinositol 3-kinase/PDK1/mTORC2 and the pleckstrin homology domain to sustain malignant transformation.

Samankaltaisia teoksia

• IKBKE Protein Activates Akt Independent of Phosphatidylinositol 3-Kinase/PDK1/mTORC2 and the Pleckstrin Homology Domain to Sustain Malignant Transformation
  Tekijä: Guo, Jian-Ping, et al.
  Julkaistu: (2011)
• PDK1-SGK1 Signaling Sustains AKT-Independent mTORC1 Activation and Confers Resistance to PI3Kα Inhibition
  Tekijä: Castel, Pau, et al.
  Julkaistu: (2016)
• Disruption of the Interface between the Pleckstrin Homology (PH) and Kinase Domains of Akt Protein Is Sufficient for Hydrophobic Motif Site Phosphorylation in the Absence of mTORC2
  Tekijä: Warfel, Noel A., et al.
  Julkaistu: (2011)
• Livers with Constitutive mTORC1 Activity Resist Steatosis Independent of Feedback Suppression of Akt
  Tekijä: Kenerson, Heidi L., et al.
  Julkaistu: (2015)
• Transient activation of mTORC1 signaling in skeletal muscle is independent of Akt1 regulation
  Tekijä: Miyazaki, Mitsunori, et al.
  Julkaistu: (2020)