Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.

In normal subjects and diabetic patients, insulin suppresses whole body proteolysis suggesting that the loss of lean body mass and muscle wasting in insulinopenia is related to increased muscle protein degradation. To document how insulinopenia affects organ weights and to identify the pathway for a...

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Бібліографічні деталі
Опубліковано в::	J Clin Invest
Автори:	Price, S R, Bailey, J L, Wang, X, Jurkovitz, C, England, B K, Ding, X, Phillips, L S, Mitch, W E
Формат:	Artigo
Мова:	Inglês
Опубліковано:	American Society for Clinical Investigation 1996
Предмети:	Research Article
Онлайн доступ:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC507607/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8878419/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI118968
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Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.

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