Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.

Samankaltaisia teoksia

• The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway.
  Tekijä: Bailey, J L, et al.
  Julkaistu: (1996)
• Ubiquitin, Proteasomes and Proteolytic Mechanisms Activated by Kidney Disease
  Tekijä: Rajan, Vik, et al.
  Julkaistu: (2008)
• Protein degradation and increased mRNAs encoding proteins of the ubiquitin-proteasome proteolytic pathway in BC3H1 myocytes require an interaction between glucocorticoids and acidification.
  Tekijä: Isozaki, U, et al.
  Julkaistu: (1996)
• Muscle wasting in a rat model of long-lasting sepsis results from the activation of lysosomal, Ca2+ -activated, and ubiquitin-proteasome proteolytic pathways.
  Tekijä: Voisin, L, et al.
  Julkaistu: (1996)
• Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes.
  Tekijä: Mitch, W E, et al.
  Julkaistu: (1994)