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Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease.
The pathogenesis of systemic lupus erythematosus (SLE) is multifactorial and multigenetic. The apoptosis genes, fas and fas ligand (fasL), are candidate contributory genes in human SLE, as mutations of these genes result in autoimmunity in several murine models of this disease. In humans, fas mutati...
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| Publicado no: | J Clin Invest |
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| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Clinical Investigation
1996
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC507531/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8787672/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI118892 |
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