Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease.

The pathogenesis of systemic lupus erythematosus (SLE) is multifactorial and multigenetic. The apoptosis genes, fas and fas ligand (fasL), are candidate contributory genes in human SLE, as mutations of these genes result in autoimmunity in several murine models of this disease. In humans, fas mutati...

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Bibliografiske detaljer

Udgivet i:	J Clin Invest
Principais autores:	Wu, J, Wilson, J, He, J, Xiang, L, Schur, P H, Mountz, J D
Format:	Artigo
Sprog:	Inglês
Udgivet:	American Society for Clinical Investigation 1996
Fag:	Research Article
Online adgang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC507531/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8787672/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI118892
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