Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease.

The pathogenesis of systemic lupus erythematosus (SLE) is multifactorial and multigenetic. The apoptosis genes, fas and fas ligand (fasL), are candidate contributory genes in human SLE, as mutations of these genes result in autoimmunity in several murine models of this disease. In humans, fas mutati...

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Detalles Bibliográficos

Publicado en:	J Clin Invest
Principais autores:	Wu, J, Wilson, J, He, J, Xiang, L, Schur, P H, Mountz, J D
Formato:	Artigo
Idioma:	Inglês
Publicado:	American Society for Clinical Investigation 1996
Assuntos:	Research Article
Acceso en liña:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC507531/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8787672/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI118892
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