ロード中...
Oxidant stress promotes disease by activating CaMKII
CaMKII is activated by oxidation of methionine residues residing in the regulatory domain. Oxidized CaMKII (ox-CaMKII) is now thought to participate in cardiovascular and pulmonary diseases and cancer. This invited review summarizes current evidence for the role of ox-CaMKII in disease, considers cr...
保存先:
| 出版年: | J Mol Cell Cardiol |
|---|---|
| 第一著者: | |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2015
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5075238/ https://ncbi.nlm.nih.gov/pubmed/26475411 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2015.10.014 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|