Loss of endogenous thymosin β(4) accelerates glomerular disease

Glomerular disease is characterized by morphologic changes in podocyte cells accompanied by inflammation and fibrosis. Thymosin β(4) regulates cell morphology, inflammation, and fibrosis in several organs and administration of exogenous thymosin β(4) improves animal models of unilateral ureteral obs...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Kidney Int
Päätekijät: Vasilopoulou, Elisavet, Kolatsi-Joannou, Maria, Lindenmeyer, Maja T., White, Kathryn E., Robson, Michael G., Cohen, Clemens D., Sebire, Neil J., Riley, Paul R., Winyard, Paul J., Long, David A.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Elsevier 2016
Aiheet:
Basic Research
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5073078/
https://ncbi.nlm.nih.gov/pubmed/27575556
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.kint.2016.06.032
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