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Loss of endogenous thymosin β(4) accelerates glomerular disease
Glomerular disease is characterized by morphologic changes in podocyte cells accompanied by inflammation and fibrosis. Thymosin β(4) regulates cell morphology, inflammation, and fibrosis in several organs and administration of exogenous thymosin β(4) improves animal models of unilateral ureteral obs...
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| Vydáno v: | Kidney Int |
|---|---|
| Hlavní autoři: | , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Elsevier
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5073078/ https://ncbi.nlm.nih.gov/pubmed/27575556 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.kint.2016.06.032 |
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