High expression of human beta S- and alpha-globins in transgenic mice: erythrocyte abnormalities, organ damage, and the effect of hypoxia.

Eitemau Tebyg

• High expression of human beta S- and alpha-globins in transgenic mice: hemoglobin composition and hematological consequences.
  gan: Fabry, M E, et al.
  Cyhoeddwyd: (1992)
• Magnetic resonance evidence of hypoxia in a homozygous alpha-knockout of a transgenic mouse model for sickle cell disease.
  gan: Fabry, M E, et al.
  Cyhoeddwyd: (1996)
• Upstream G gamma-globin and downstream beta-globin sequences required for stage-specific expression in transgenic mice.
  gan: Trudel, M, et al.
  Cyhoeddwyd: (1987)
• Beta-globin enhancers target expression of a heterologous gene to erythroid tissues of transgenic mice.
  gan: Magram, J, et al.
  Cyhoeddwyd: (1989)
• Abnormal splice in a mutant human beta-globin gene not at the site of a mutation.
  gan: Dobkin, C, et al.
  Cyhoeddwyd: (1983)