Hyperosmotic stress induces Rho/Rho kinase/LIM kinase-mediated cofilin phosphorylation in tubular cells: key role in the osmotically triggered F-actin response

Hyperosmotic stress induces cytoskeleton reorganization and a net increase in cellular F-actin, but the underlying mechanisms are incompletely understood. Whereas de novo F-actin polymerization likely contributes to the actin response, the role of F-actin severing is unknown. To address this problem...

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Gepubliceerd in:Am J Physiol Cell Physiol
Hoofdauteurs: Thirone, Ana C. P., Speight, Pam, Zulys, Matthew, Rotstein, Ori D., Szászi, Katalin, Pedersen, Stine F., Kapus, András
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2008
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5047760/
https://ncbi.nlm.nih.gov/pubmed/19109524
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00467.2008
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