p53 and p16(Ink4a)/p19(Arf) Loss Promotes Different Pancreatic Tumor Types from PyMT-Expressing Progenitor Cells()()

In human studies and mouse models, the contributions of p53 and p16(Ink4a)/p19(Arf) loss are well established in pancreatic ductal adenocarcinoma (PDAC). Although loss of functional p53 pathway and loss of Ink4a/Arf in human pancreatic acinar cell carcinoma (PACC) and pancreatic neuroendocrine tumor...

Description complète

Enregistré dans:
Détails bibliographiques
Publié dans:Neoplasia
Auteurs principaux: Azzopardi, Stephanie, Pang, Sharon, Klimstra, David S., Du, Yi-Chieh Nancy
Format: Artigo
Langue:Inglês
Publié: Neoplasia Press 2016
Sujets:
Original article
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5035259/
https://ncbi.nlm.nih.gov/pubmed/27664376
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neo.2016.08.003
Tags: Ajouter un tag
Pas de tags, Soyez le premier à ajouter un tag!

Documents similaires