p53 and p16(Ink4a)/p19(Arf) Loss Promotes Different Pancreatic Tumor Types from PyMT-Expressing Progenitor Cells()()

In human studies and mouse models, the contributions of p53 and p16(Ink4a)/p19(Arf) loss are well established in pancreatic ductal adenocarcinoma (PDAC). Although loss of functional p53 pathway and loss of Ink4a/Arf in human pancreatic acinar cell carcinoma (PACC) and pancreatic neuroendocrine tumor...

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Publicat a:Neoplasia
Autors principals: Azzopardi, Stephanie, Pang, Sharon, Klimstra, David S., Du, Yi-Chieh Nancy
Format: Artigo
Idioma:Inglês
Publicat: Neoplasia Press 2016
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Original article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5035259/
https://ncbi.nlm.nih.gov/pubmed/27664376
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neo.2016.08.003
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