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New Developments in Anti-Sickling Agents: Can Drugs Directly Prevent the Polymerization of Sickle Haemoglobin In Vivo?

The hallmark of sickle cell disease is the polymerization of sickle haemoglobin due to a point mutation in the β-globin gene (HBB). Under low oxygen saturation, sickle haemoglobin assumes the tense (T-state) deoxygenated conformation that can form polymers, leading to rigid erythrocytes with impaire...

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Bibliografiska uppgifter
I publikationen:Br J Haematol
Huvudupphovsmän: Oder, Esther, Safo, Martin K., Abdulmalik, Osheiza, Kato, Gregory J.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2016
Ämnen:
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC5035193/
https://ncbi.nlm.nih.gov/pubmed/27605087
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/bjh.14264
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