Complement activation by beta-amyloid in Alzheimer disease.

Alzheimer disease (AD) is characterized by excessive deposition of the beta-amyloid peptide (beta-AP) in the central nervous system. Although several lines of evidence suggest that beta-AP is neurotoxic, a mechanism for beta-AP toxicity in AD brain remains unclear. In this paper we provide both dire...

Полное описание

Библиографические подробности

Опубликовано в::	Proc Natl Acad Sci U S A
Главные авторы:	Rogers, J, Cooper, N R, Webster, S, Schultz, J, McGeer, P L, Styren, S D, Civin, W H, Brachova, L, Bradt, B, Ward, P
Формат:	Artigo
Язык:	Inglês
Опубликовано:	National Academy of Sciences 1992
Предметы:	Research Article
Online-ссылка:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC50268/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1438191/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.89.21.10016
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