Complement activation by beta-amyloid in Alzheimer disease.

Alzheimer disease (AD) is characterized by excessive deposition of the beta-amyloid peptide (beta-AP) in the central nervous system. Although several lines of evidence suggest that beta-AP is neurotoxic, a mechanism for beta-AP toxicity in AD brain remains unclear. In this paper we provide both dire...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Rogers, J, Cooper, N R, Webster, S, Schultz, J, McGeer, P L, Styren, S D, Civin, W H, Brachova, L, Bradt, B, Ward, P
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 1992
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC50268/
https://ncbi.nlm.nih.gov/pubmed/1438191
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