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Amyloid-β42 protofibrils are internalized by microglia more extensively than monomers
One pathological hallmark of Alzheimer’s disease (AD) is the accumulation of amyloid-β peptide (Aβ) in the affected brain. While there are numerous deleterious effects of Aβ accumulation, there is general agreement that a sustained inflammatory response to aggregated Aβ contributes to progressive ne...
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| Publicado no: | Brain Res |
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| Principais autores: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5018462/ https://ncbi.nlm.nih.gov/pubmed/27531183 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2016.08.016 |
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