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The combination of vemurafenib and procaspase-3 activation is synergistic in mutant BRAF melanomas

The development of vemurafenib resistance limits the long-term efficacy of this drug for treatment of metastatic melanomas with the (V600E)BRAF mutation. Inhibition of downstream MAPK signaling with vemurafenib induces apoptotic cell death mediated by caspase-3, suggesting that addition of a procasp...

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Publicat a:Mol Cancer Ther
Autors principals: Peh, Jessie, Fan, Timothy M., Wycislo, Kathryn L., Roth, Howard S., Hergenrother, Paul J.
Format: Artigo
Idioma:Inglês
Publicat: 2016
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4975653/
https://ncbi.nlm.nih.gov/pubmed/27297867
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/1535-7163.MCT-16-0025
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