Developmental acceleration of bradykinin-dependent relaxation by prenatal chronic hypoxia impedes normal development after birth

Bradykinin-induced activation of the pulmonary endothelium triggers nitric oxide production and other signals that cause vasorelaxation, including stimulation of large-conductance Ca(2+)-activated K(+) (BK(Ca)) channels in myocytes that hyperpolarize the plasma membrane and decrease intracellular Ca...

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Veröffentlicht in:Am J Physiol Lung Cell Mol Physiol
Hauptverfasser: Blum-Johnston, Carla, Thorpe, Richard B., Wee, Chelsea, Romero, Monica, Brunelle, Alexander, Blood, Quintin, Wilson, Rachael, Blood, Arlin B., Francis, Michael, Taylor, Mark S., Longo, Lawrence D., Pearce, William J., Wilson, Sean M.
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Physiological Society 2015
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Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4971892/
https://ncbi.nlm.nih.gov/pubmed/26637638
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00340.2015
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