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Developmental acceleration of bradykinin-dependent relaxation by prenatal chronic hypoxia impedes normal development after birth
Bradykinin-induced activation of the pulmonary endothelium triggers nitric oxide production and other signals that cause vasorelaxation, including stimulation of large-conductance Ca(2+)-activated K(+) (BK(Ca)) channels in myocytes that hyperpolarize the plasma membrane and decrease intracellular Ca...
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| Publicado no: | Am J Physiol Lung Cell Mol Physiol |
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| Main Authors: | , , , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4971892/ https://ncbi.nlm.nih.gov/pubmed/26637638 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00340.2015 |
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