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MMP-13 is one of the critical mediators of the effect of HDAC4 deletion on the skeleton
Histone deacetylase 4 (Hdac4) regulates chondrocyte hypertrophy. Hdac4(−/−) mice are runted in size and do not survive to weaning. This phenotype is primarily due to the acceleration of onset of chondrocyte hypertrophy and, as a consequence, inappropriate endochondral mineralization. Previously, we...
Tallennettuna:
| Julkaisussa: | Bone |
|---|---|
| Päätekijät: | , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2016
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4970950/ https://ncbi.nlm.nih.gov/pubmed/27320207 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bone.2016.06.010 |
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