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The deletion of Hdac4 in mouse osteoblasts influences both catabolic and anabolic effects in bone
Histone deacetylase 4 (Hdac4) is known to control chondrocyte hypertrophy and bone formation. We have previously shown that parathyroid hormone (PTH) regulates many aspects of Hdac4 function in osteoblastic cells in vitro; however, in vivo confirmation was previously precluded by pre-weaning lethali...
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| Publicado no: | J Bone Miner Res |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2018
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6457245/ https://ncbi.nlm.nih.gov/pubmed/29544022 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jbmr.3422 |
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