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SCF(Fbl12) Increases p21(Waf1/Cip1) Expression Level through Atypical Ubiquitin Chain Synthesis
The cyclin-dependent kinase (CDK) inhibitor p21 is an unstructured protein regulated by multiple turnover pathways. p21 abundance is tightly regulated, and its defect causes tumor development. However, the mechanisms that underlie the control of p21 level are not fully understood. Here, we report a...
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| 出版年: | Mol Cell Biol |
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| 主要な著者: | , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4968210/ https://ncbi.nlm.nih.gov/pubmed/27215384 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.00174-16 |
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