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Cyclic ADP-Ribose and Heat Regulate Oxytocin Release via CD38 and TRPM2 in the Hypothalamus during Social or Psychological Stress in Mice
Hypothalamic oxytocin (OT) is released into the brain by cyclic ADP-ribose (cADPR) with or without depolarizing stimulation. Previously, we showed that the intracellular free calcium concentration ([Ca(2+)](i)) that seems to trigger OT release can be elevated by β-NAD(+), cADPR, and ADP in mouse oxy...
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| Udgivet i: | Front Neurosci |
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| Main Authors: | , , , , , , , , , , , , , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Frontiers Media S.A.
2016
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4956647/ https://ncbi.nlm.nih.gov/pubmed/27499729 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fnins.2016.00304 |
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