Gestational Diabetes Mellitus From Inactivation of Prolactin Receptor and MafB in Islet β-Cells

β-Cell proliferation and expansion during pregnancy are crucial for maintaining euglycemia in response to increased metabolic demands placed on the mother. Prolactin and placental lactogen signal through the prolactin receptor (PRLR) and contribute to adaptive β-cell responses in pregnancy; however,...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Diabetes
Päätekijät: Banerjee, Ronadip R., Cyphert, Holly A., Walker, Emily M., Chakravarthy, Harini, Peiris, Heshan, Gu, Xueying, Liu, Yinghua, Conrad, Elizabeth, Goodrich, Lisa, Stein, Roland W., Kim, Seung K.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Diabetes Association 2016
Aiheet:
Islet Studies
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4955982/
https://ncbi.nlm.nih.gov/pubmed/27217483
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2337/db15-1527
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