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Ceapins inhibit ATF6α signaling by selectively preventing transport of ATF6α to the Golgi apparatus during ER stress

The membrane-bound transcription factor ATF6α is activated by proteolysis during endoplasmic reticulum (ER) stress. ATF6α target genes encode foldases, chaperones, and lipid biosynthesis enzymes that increase protein-folding capacity in response to demand. The off-state of ATF6α is maintained by its...

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Bibliographic Details
Published in:eLife
Main Authors: Gallagher, Ciara M, Walter, Peter
Format: Artigo
Language:Inglês
Published: eLife Sciences Publications, Ltd 2016
Subjects:
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC4954756/
https://ncbi.nlm.nih.gov/pubmed/27435962
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.11880
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