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The DNA damage response induces inflammation and senescence by inhibiting autophagy of GATA4
Cellular senescence is a terminal stress-activated program controlled by the p53 and p16(INK4a) tumor suppressor proteins. A striking feature of senescence is the senescence-associated secretory phenotype (SASP), a pro-inflammatory response linked to tumor promotion and aging. We have identified the...
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| Publicado no: | Science |
|---|---|
| Main Authors: | , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4942138/ https://ncbi.nlm.nih.gov/pubmed/26404840 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/science.aaa5612 |
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