Targeted overexpression of endothelial nitric oxide synthase in endothelial cells improves cerebrovascular reactivity in Ins2(Akita)-type-1 diabetic mice

Reduced bioavailability of nitric oxide due to impaired endothelial nitric oxide synthase (eNOS) activity is a leading cause of endothelial dysfunction in diabetes. Enhancing eNOS activity in diabetes is a potential therapeutic target. This study investigated basal cerebral blood flow and cerebrovas...

Descripció completa

Guardat en:
Dades bibliogràfiques
Publicat a:J Cereb Blood Flow Metab
Autors principals: Chandra, Saurav B, Mohan, Sumathy, Ford, Bridget M, Huang, Lei, Janardhanan, Preethi, Deo, Kaiwalya S, Cong, Linlin, Muir, Eric R, Duong, Timothy Q
Format: Artigo
Idioma:Inglês
Publicat: SAGE Publications 2015
Matèries:
Original Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4908624/
https://ncbi.nlm.nih.gov/pubmed/26661212
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X15612098
Etiquetes: Afegir etiqueta
Sense etiquetes, Sigues el primer a etiquetar aquest registre!

Ítems similars