Targeted overexpression of endothelial nitric oxide synthase in endothelial cells improves cerebrovascular reactivity in Ins2(Akita)-type-1 diabetic mice

Reduced bioavailability of nitric oxide due to impaired endothelial nitric oxide synthase (eNOS) activity is a leading cause of endothelial dysfunction in diabetes. Enhancing eNOS activity in diabetes is a potential therapeutic target. This study investigated basal cerebral blood flow and cerebrovas...

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Dettagli Bibliografici
Pubblicato in:J Cereb Blood Flow Metab
Autori principali: Chandra, Saurav B, Mohan, Sumathy, Ford, Bridget M, Huang, Lei, Janardhanan, Preethi, Deo, Kaiwalya S, Cong, Linlin, Muir, Eric R, Duong, Timothy Q
Natura: Artigo
Lingua:Inglês
Pubblicazione: SAGE Publications 2015
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Original Articles
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4908624/
https://ncbi.nlm.nih.gov/pubmed/26661212
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X15612098
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