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Alveolar epithelial cell-derived prostaglandin E(2) serves as a request signal for macrophage secretion of suppressor of cytokine signaling 3 during innate inflammation
Preservation of gas exchange mandates that the pulmonary alveolar surface restrain unnecessarily harmful inflammatory responses to the many challenges to which it is exposed. These responses reflect the crosstalk between alveolar epithelial cells (AECs) and resident alveolar macrophages (AMs). We re...
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| Publicado no: | J Immunol |
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| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4893973/ https://ncbi.nlm.nih.gov/pubmed/27183597 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1502153 |
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