טוען...
IL-17A deficiency promotes periosteal bone formation in a model of inflammatory arthritis
BACKGROUND: Interleukin-17A (IL-17A) plays a pathogenic role in several rheumatic diseases including spondyloarthritis and, paradoxically, has been described to both promote and protect from bone formation. We therefore examined the effects of IL-17A on osteoblast differentiation in vitro and on per...
שמור ב:
| הוצא לאור ב: | Arthritis Res Ther |
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| Main Authors: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
BioMed Central
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4863346/ https://ncbi.nlm.nih.gov/pubmed/27165410 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13075-016-0998-x |
| תגים: |
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