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ANP32B deficiency impairs proliferation and suppresses tumor progression by regulating AKT phosphorylation
The acidic leucine-rich nuclear phosphoprotein 32B (ANP32B) is reported to impact normal development, with Anp32b-knockout mice exhibiting smaller size and premature aging. However, its cellular and molecular mechanisms, especially its potential roles in tumorigenesis, remain largely unclear. Here,...
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| 出版年: | Cell Death Dis |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4849165/ https://ncbi.nlm.nih.gov/pubmed/26844697 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2016.8 |
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