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Growth hormone (GH)‐transgenic insulin‐like growth factor 1 (IGF1)‐deficient mice allow dissociation of excess GH and IGF1 effects on glomerular and tubular growth

Growth hormone (GH)‐transgenic mice with permanently elevated systemic levels of GH and insulin‐like growth factor 1 (IGF1) reproducibly develop renal and glomerular hypertrophy and subsequent progressive glomerulosclerosis, finally leading to terminal renal failure. To dissociate IGF1‐dependent and...

詳細記述

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書誌詳細
出版年:Physiol Rep
主要な著者: Blutke, Andreas, Schneider, Marlon R., Wolf, Eckhard, Wanke, Rüdiger
フォーマット: Artigo
言語:Inglês
出版事項: John Wiley and Sons Inc. 2016
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC4823598/
https://ncbi.nlm.nih.gov/pubmed/26997624
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.12709
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