Connexin 32 dysfunction promotes ethanol-related hepatocarcinogenesis via activation of Dusp1-Erk axis

There is abundant epidemiological evidence that heavy alcohol intake contributes to hepatocellular carcinoma (HCC) development. Previous reports indicated that connexin 32 (Cx32), which is a major hepatocyte gap junction protein, is down-regulated in chronic liver disease and has a protective role i...

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Shranjeno v:
Bibliografske podrobnosti
izdano v:Oncotarget
Main Authors: Kato, Hiroyuki, Naiki-Ito, Aya, Naiki, Taku, Suzuki, Shugo, Yamashita, Yoriko, Sato, Shinya, Sagawa, Hiroyuki, Kato, Akihisa, Kuno, Toshiya, Takahashi, Satoru
Format: Artigo
Jezik:Inglês
Izdano: Impact Journals LLC 2015
Teme:
Research Paper
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4811513/
https://ncbi.nlm.nih.gov/pubmed/26655499
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