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Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth
BACKGROUND: Neo-angiogenesis is a hallmark of glioblastoma (GBM) and is sustained by autocrine and paracrine interactions between neoplastic and nonneoplastic cells. Programmed cell death 10 (PDCD10) is ubiquitously expressed in nearly all tissues and plays crucial roles in regulating angiogenesis a...
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| 出版年: | Neuro Oncol |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Oxford University Press
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4799675/ https://ncbi.nlm.nih.gov/pubmed/26254477 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/nov155 |
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