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Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth
BACKGROUND: Neo-angiogenesis is a hallmark of glioblastoma (GBM) and is sustained by autocrine and paracrine interactions between neoplastic and nonneoplastic cells. Programmed cell death 10 (PDCD10) is ubiquitously expressed in nearly all tissues and plays crucial roles in regulating angiogenesis a...
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| Publicado no: | Neuro Oncol |
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| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4799675/ https://ncbi.nlm.nih.gov/pubmed/26254477 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/nov155 |
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