Excitatory transmission onto AgRP neurons is regulated by cJun NH(2)-terminal kinase 3 in response to metabolic stress

The cJun NH(2)-terminal kinase (JNK) signaling pathway is implicated in the response to metabolic stress. Indeed, it is established that the ubiquitously expressed JNK1 and JNK2 isoforms regulate energy expenditure and insulin resistance. However, the role of the neuron-specific isoform JNK3 is uncl...

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Kaydedildi:
Detaylı Bibliyografya
Yayımlandı:eLife
Asıl Yazarlar: Vernia, Santiago, Morel, Caroline, Madara, Joseph C, Cavanagh-Kyros, Julie, Barrett, Tamera, Chase, Kathryn, Kennedy, Norman J, Jung, Dae Young, Kim, Jason K, Aronin, Neil, Flavell, Richard A, Lowell, Bradford B, Davis, Roger J
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: eLife Sciences Publications, Ltd 2016
Konular:
Cell Biology
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4798947/
https://ncbi.nlm.nih.gov/pubmed/26910012
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.10031
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