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Neutrophil effector responses are suppressed by secretory phospholipase A(2) modified HDL
Secretory phospholipase A(2) (sPLA(2)) generates bioactive lysophospholipids implicated in acute and chronic inflammation, but the pathophysiologic role of sPLA(2) is poorly understood. Given that high-density lipoprotein (HDL) is the major substrate for sPLA(2) in plasma, we investigated the effect...
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出版年: | Biochim Biophys Acta |
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主要な著者: | , , , , , , |
フォーマット: | Artigo |
言語: | Inglês |
出版事項: |
2014
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主題: | |
オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4789498/ https://ncbi.nlm.nih.gov/pubmed/25463476 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbalip.2014.11.010 |
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