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Neutrophil effector responses are suppressed by secretory phospholipase A(2) modified HDL

Secretory phospholipase A(2) (sPLA(2)) generates bioactive lysophospholipids implicated in acute and chronic inflammation, but the pathophysiologic role of sPLA(2) is poorly understood. Given that high-density lipoprotein (HDL) is the major substrate for sPLA(2) in plasma, we investigated the effect...

詳細記述

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書誌詳細
出版年:Biochim Biophys Acta
主要な著者: Curcic, Sanja, Holzer, Michael, Frei, Robert, Pasterk, Lisa, Schicho, Rudolf, Heinemann, Akos, Marsche, Gunther
フォーマット: Artigo
言語:Inglês
出版事項: 2014
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC4789498/
https://ncbi.nlm.nih.gov/pubmed/25463476
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbalip.2014.11.010
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