Absence of Prenatal Forebrain Defects in the Dp(16)1Yey/+ Mouse Model of Down Syndrome

Studies in humans with Down syndrome (DS) show that alterations in fetal brain development are followed by postnatal deficits in neuronal numbers, synaptic plasticity, and cognitive and motor function. This same progression is replicated in several mouse models of DS. Dp(16)1Yey/+ (hereafter called...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Goodliffe, Joseph W., Olmos-Serrano, Jose Luis, Aziz, Nadine M., Pennings, Jeroen L.A., Guedj, Faycal, Bianchi, Diana W., Haydar, Tarik F.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2016
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783496/
https://ncbi.nlm.nih.gov/pubmed/26961948
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.2513-15.2016
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