Absence of Prenatal Forebrain Defects in the Dp(16)1Yey/+ Mouse Model of Down Syndrome

Studies in humans with Down syndrome (DS) show that alterations in fetal brain development are followed by postnatal deficits in neuronal numbers, synaptic plasticity, and cognitive and motor function. This same progression is replicated in several mouse models of DS. Dp(16)1Yey/+ (hereafter called...

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Bibliografske podrobnosti
izdano v:J Neurosci
Main Authors: Goodliffe, Joseph W., Olmos-Serrano, Jose Luis, Aziz, Nadine M., Pennings, Jeroen L.A., Guedj, Faycal, Bianchi, Diana W., Haydar, Tarik F.
Format: Artigo
Jezik:Inglês
Izdano: Society for Neuroscience 2016
Teme:
Articles
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783496/
https://ncbi.nlm.nih.gov/pubmed/26961948
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.2513-15.2016
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