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Loss of OMA1 delays neurodegeneration by preventing stress-induced OPA1 processing in mitochondria
Proteolytic cleavage of the dynamin-like guanosine triphosphatase OPA1 in mitochondria is emerging as a central regulatory hub that determines mitochondrial morphology under stress and in disease. Stress-induced OPA1 processing by OMA1 triggersmitochondrial fragmentation, which is associated with mi...
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| Publicado no: | J Cell Biol |
|---|---|
| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The Rockefeller University Press
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4738383/ https://ncbi.nlm.nih.gov/pubmed/26783299 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.201507022 |
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