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Lack of hepcidin expression attenuates steatosis and causes fibrosis in the liver
AIM: To investigate the role of key iron-regulatory protein, hepcidin in non-alcoholic fatty liver disease (NAFLD). METHODS: Hepcidin (Hamp1) knockout and floxed control mice were administered a high fat and high sucrose (HFS) or a regular control diet for 3 or 7 mo. Steatosis, triglycerides, fibros...
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| Vydáno v: | World J Hepatol |
|---|---|
| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Baishideng Publishing Group Inc
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4733464/ https://ncbi.nlm.nih.gov/pubmed/26855692 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4254/wjh.v8.i4.211 |
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