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Fucosylation and protein glycosylation create functional receptors for cholera toxin
Cholera toxin (CT) enters and intoxicates host cells after binding cell surface receptors using its B subunit (CTB). The ganglioside (glycolipid) GM1 is thought to be the sole CT receptor; however, the mechanism by which CTB binding to GM1 mediates internalization of CT remains enigmatic. Here we re...
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| 出版年: | eLife |
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| 主要な著者: | , , , , , , , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
eLife Sciences Publications, Ltd
2015
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4686427/ https://ncbi.nlm.nih.gov/pubmed/26512888 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.09545 |
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