The point mutation UCH-L1 C152A protects primary neurons against cyclopentenone prostaglandin-induced cytotoxicity: implications for post-ischemic neuronal injury

Lignende værker

• Protein disulfide isomerase as a novel target for cyclopentenone prostaglandins: implications for hypoxic ischemic injury
  af: Liu, Hao, et al.
  Udgivet: (2015)
• Modification of ubiquitin-C-terminal hydrolase-L1 by cyclopentenone prostaglandins exacerbates hypoxic injury
  af: Liu, Hao, et al.
  Udgivet: (2010)
• Cyclopentenone prostaglandin-induced unfolding and aggregation of the Parkinson disease-associated UCH-L1
  af: Koharudin, Leonardus M. I., et al.
  Udgivet: (2010)
• Prostaglandin D(2) toxicity in primary neurons is mediated through its bioactive cyclopentenone metabolites
  af: Liu, Hao, et al.
  Udgivet: (2013)
• Increased generation of cyclopentenone prostaglandins after brain ischemia and their role in aggregation of ubiquitinated proteins in neurons
  af: Liu, Hao, et al.
  Udgivet: (2013)