S-Nitrosylation of Sarcomeric Proteins Depresses Myofilament Ca(2+) Sensitivity in Intact Cardiomyocytes

Aims: The heart responds to physiological and pathophysiological stress factors by increasing its production of nitric oxide (NO), which reacts with intracellular glutathione to form S-nitrosoglutathione (GSNO), a protein S-nitrosylating agent. Although S-nitrosylation protects some cardiac proteins...

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Dades bibliogràfiques
Publicat a:Antioxid Redox Signal
Autors principals: Figueiredo-Freitas, Cícero, Dulce, Raul A., Foster, Matthew W., Liang, Jingsheng, Yamashita, Aline M.S., Lima-Rosa, Frederico L., Thompson, J. Will, Moseley, M. Arthur, Hare, Joshua M., Nogueira, Leonardo, Sorenson, Martha M., Pinto, José Renato
Format: Artigo
Idioma:Inglês
Publicat: Mary Ann Liebert, Inc. 2015
Matèries:
Original Research Communications
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4649751/
https://ncbi.nlm.nih.gov/pubmed/26421519
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ars.2015.6275
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