S-phase-dependent p50/NF-кB1 phosphorylation in response to ATR and replication stress acts to maintain genomic stability

The apical damage kinase, ATR, is activated by replication stress (RS) both in response to DNA damage and during normal S-phase. Loss of function studies indicates that ATR acts to stabilize replication forks, block cell cycle progression and promote replication restart. Although checkpoint failure...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Cell Cycle
Egile Nagusiak: Crawley, Clayton D, Kang, Shijun, Bernal, Giovanna M, Wahlstrom, Joshua S, Voce, David J, Cahill, Kirk E, Garofalo, Andrea, Raleigh, David R, Weichselbaum, Ralph R, Yamini, Bakhtiar
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Taylor & Francis 2015
Gaiak:
Reports
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC4614791/
https://ncbi.nlm.nih.gov/pubmed/25590437
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/15384101.2014.991166
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