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Repair of persistent strand breaks in the mitochondrial genome
Oxidative DNA damage has been attributed to increased cancer incidence and premature aging phenotypes. Reactive oxygen species (ROS) are unavoidable byproducts of oxidative phosphorylation and are the major contributors of endogenous oxidative damage. To prevent the negative effects of ROS, cells ha...
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| 發表在: | Mech Ageing Dev |
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| Main Authors: | , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
2011
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4586262/ https://ncbi.nlm.nih.gov/pubmed/22138376 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mad.2011.11.003 |
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