Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis and lipogenic pathways in a mature rat model of non-alcoholic fatty-liver disease

Nonalcoholic fatty-liver disease (NAFLD) prevalence is increasing worldwide, with the affected US population estimated near 30%. Diet is a recognized risk factor in the NAFLD spectrum, which includes non-alcoholic steatohepatitis (NASH) and fibrosis. Low hepatic copper (Cu) was recently linked to cl...

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Veröffentlicht in:J Nutr Biochem
Hauptverfasser: Tallino, Savannah, Duffy, Megan, Ralle, Martina, Cortés, María Paz, Latorre, Mauricio, Burkhead, Jason L.
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2015
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4578989/
https://ncbi.nlm.nih.gov/pubmed/26033743
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jnutbio.2015.04.009
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