Genetic Deletion of β-Arrestin-2 and the Mitigation of Established Airway Hyperresponsiveness in a Murine Asthma Model

β-Arrestin-2 (βarr2) is a ubiquitously expressed cytosolic protein that terminates G protein–coupled receptor signaling and transduces G protein–independent signaling. We previously showed that mice lacking βarr2 do not develop an asthma phenotype when sensitized to, and challenged with, allergens....

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Publicat a:Am J Respir Cell Mol Biol
Autors principals: Chen, Minyong, Hegde, Akhil, Choi, Yeon Ho, Theriot, Barbara S., Premont, Richard T., Chen, Wei, Walker, Julia K. L.
Format: Artigo
Idioma:Inglês
Publicat: American Thoracic Society 2015
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Original Research
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4566063/
https://ncbi.nlm.nih.gov/pubmed/25569510
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1165/rcmb.2014-0231OC
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