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FAK regulates cardiomyocyte survival following ischemia/reperfusion
Myocyte apoptosis is central to myocardial dysfunction following ischemia/reperfusion (I/R) and during the transition from hypertrophy to heart failure. Focal adhesion kinase (FAK), a non-receptor tyrosine kinase regulates adhesion-dependent survival signals and unopposed FAK activation has been lin...
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| Izdano u: | J Mol Cell Cardiol |
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| Glavni autori: | , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2008
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4535182/ https://ncbi.nlm.nih.gov/pubmed/19028502 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2008.10.017 |
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