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Mutation of a Src phosphorylation site in the PDGF beta-receptor leads to increased PDGF-stimulated chemotaxis but decreased mitogenesis.

Ligand induced activation of the beta-receptor for platelet-derived growth factor (PDGF) leads to activation of Src family tyrosine kinases. We have explored the possibility that the receptor itself is a substrate for Src. We show that Tyr934 in the kinase domain of the PDGF receptor is phosphorylat...

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Wedi'i Gadw mewn:
Cyhoeddwyd yn:EMBO J
Prif Awduron: Hansen, K, Johnell, M, Siegbahn, A, Rorsman, C, Engström, U, Wernstedt, C, Heldin, C H, Rönnstrand, L
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: Nature Publishing Group 1996
Pynciau:
Research Article
Mynediad Ar-lein:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC452274/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8895575/
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